By C. M. Grondin, J. C. Thornton (auth.), Professor Dr. med. Thomas F. Lüscher, Professor Dr. med. Marko Turina, Professor Dr. med. Eugene Braunwald (eds.)
In sufferers with coronary artery sickness, surgical revascu- larization with arterial or venous pass grafts not just relieves signs, but additionally prolongs lifestyles. the results of such interventions, even if, is usually impaired through graft disorder and occlusion. This monograph highlights the scientific significance of coronary artery skip graft sickness and, specifically, using glossy diagnostic suggestions to evaluate graft constitution and serve as. The molecular and mobile mechanisms of coronary skip graft illness are ex- tensively mentioned with numerous chapters dedicated to prophy- lactic clinical remedy. The indication, method and result of reinterventions with balloon angioplasty, reope- ration or transplantation in sufferers with graft failure also are reviewed.
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Additional resources for Coronary Artery Graft Disease: Mechanisms and Prevention
Saunders, Philadelphia, pp 375-388 13. Chesebro JH, Clements IP, Fuster V, Elveback LR, Smith HC, Bardsley WT, Frye RL, Homes DR, Vlietstra RE, Pluth JR, Wallace RB, Puga FJ, Orszulak TA, Piehler JM, Schaff HV, Danielson GK (1982) A platelet-inhibitor-drug trial in coronary-artery bypass operations: benefit of perioperative dipyridamole and aspirin therapy on early postoperative vein-graft patency. N Engl J Med 307:73-78 14. Clowes AW, Schwartz SM (1985) Significance of quiescent smooth muscle migration in the injured rat carotid artery.
2). Tissue factor activates not only the intrinsic pathway but also the extrinsic pathway by stimulating factor IX in the presence of calcium. The final product, thrombin, catalyzes fibrinogen to fibrin, activates factor XIII to promote cross-linking of fibrin, and produces new thrombin by stimulating factors V and VIII. At the same Mechanisms of Plaque Formation and Occlusion in Venous Coronary Bypass Grafts 43 EVOLUTION AND PROGRESSION OF ATHEROSCLEROSIS VEIN GRAFT ATHEROSCLEROSIS 3 months Endothelium Intima Media Adventitia 1 year 3 - 6 years r----- Type I .
J BioI Chern 265:17334-17440 5. Bini A, Fenoglio JJ, Mes-Tejada R, Kerdryk B, Kaplan KL (1989) Identification and distribution of fibrinogen, fibrin and fibrin (ogen) degradation products in atherosclerosis: use of monoclonal antibodies. Atherosclerosis 1:109-121 6. Bulkley BH, Hutchins GM (1978) Pathology of coronary artery bypass graft surgery. Arch Pathol Lab Med 102:273-280 7. Campbell GR, Campbell JH (1985) Smooth muscle phenotypic changes in arterial wall homeostasis: implication for the pathogenesis of atherosclerosis.
Coronary Artery Graft Disease: Mechanisms and Prevention by C. M. Grondin, J. C. Thornton (auth.), Professor Dr. med. Thomas F. Lüscher, Professor Dr. med. Marko Turina, Professor Dr. med. Eugene Braunwald (eds.)
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